In contrast, we anticipated that the reversal of this sample with delayed activation of higher airway inspiratory muscles can be associated with upper airway narrowing or collapse. We hypothesized that activation of higher airway muscle phasic electrical exercise earlier than activation of the chest wall pump muscles would assist preserve upper airway patency. Therefore the timing and amplitude of midline transmandibular and costal margin moving time average (MTA) electromyogram (EMG) signals were analyzed from 58 apnea cycles in stage 2 sleep in six OSA patients. In 86% of the postapnea breaths analyzed the higher airway MTA peak exercise preceded the chest wall peak activity. An imbalance within the amplitude of electrical activity of the higher airway and chest wall inspiratory muscles is associated with both collapse and reopening of the upper airway in obstructive sleep apnea (OSA). During inspiratory efforts when phasic inspiratory EMG amplitude did not change from preapnea to apnea, the timing modifications noted above occurred. The aim of this study was to study whether or not timing of the phasic activity of those inspiratory muscles additionally was related to modifications in upper airway caliber in OSA. Even within breaths the relative timing of the upper airway and chest wall electrical actions was closely associated with adjustments within the strain-circulate relationship. The onset of phasic electrical activity followed this same pattern. In 86% of the obstructed respiratory efforts the higher airway MTA peak exercise adopted the chest wall peak exercise.
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